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 Age-Related Macular Degeneration (ARMD)
This
is a degenerative condition of the macular. It is the most common cause of vision loss in the United States in those 50 or older, and its prevalence increases with age. Macular Degeneration (MD), is caused by hardening of the arteries
that nourish the retina. This deprives the sensitive retinal tissue of oxygen and nutrients that it needs to function and
thrive. As a result, the central vision deteriorates.
MD varies widely in severity. In the worst cases, it causes
a compete loss of central vision, making reading or driving impossible. For others, it may only cause slight distortion. Fortunately,
MD does to cause total blindness since it does not affect the peripheral vision. (As illustrated below)
What is the difference between Wet and Dry MD?
MD is classified as either wet (neovascular) or dry ( non-neovascular). About 10% of patients who suffer from MD
have wet MD. This type occures when new vessels form to improve the blood supply to exygen deprived retinal tissue.
However, the new vessels are very delicate and break easily, causing bleeding and damage to surrounding tissue. Patients with wet macular degeneration, develop new blood vessels under the retina.
This causes hemorrhages, swelling and scar tissue but it can be treated with laser in some cases. (Illustration on your right)
Dry macular degeneration, although more common, typically results
in a less severe, more gradual loss of vision. The dry type is much more common and is characterized by drusen and
loss of pigment in the retina. Drusen are small, yellow deposits that form within the layers of the retina. (Illustration
below) 
Drusen
SIGNS & SYMPTOMS *Loss of central vision (Gradual with dry MD, sudden with wet MD) *Difficulty reading or performing tasks that require detail *Distorted vision (Straight
lines such as doorways or edges of windows may appear wavy or bent.
DETECTION & DIAGNOSIS
 Vision testing, Amsler Grid, Ophthalmoscopy, Fundus Photography
and Fluorescein Angiography. Amsler Grid (You can right click an send to your printer) ____________________________________________________

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*Use a bright reading light *Wear your reading glasses
(bifocals, progressive etc) *Cover one eye *Look at center dot *Note irregularities (wavy, size, gray, fuzzy) *Repeat test with other eye *Contact your eye professional if you see any irregularities or notice any changes
TREATMENT There is no proven medical therapy for dry macular degeneration. In selected cases of
wet MD, laser photocoagulation is effective for sealing leaking or bleeding vessels.
Low Vision devices are available
for ARMD, click on our "MDE Low Vision Store" to
buy Low Vision Devices.

Amblyopia
This is a term used to describe an uncorrectable loss of vision in an eye
that appears to be normal. It's commonly referred to as lazy eye and can occur for a variety of reasons. A child's visual system is fully developed between the ages of 9-11. Until then,
children readily adapt to visual problems by suppressing or blocking out image. If caught early, the problem can often be
corrected and the vision preserved. However, after about age 11, it is difficult if not impossible to train the brain to use
the eye normally.
Some causes of amblyopia include: strabismus (croosed or tuned eye) congenital cataracts, cloudy
cornea, droopy eyelid, unequal vision and uncorrected nearsightedness, farsightedness or astigmatism. Amblyopia may occur
in various degrees depending on the severity of the underlying problem. Some patients just experience a partial loss; others
are only able to recognize motion. These patients lack binocular vision.
SIGNS AND SYMPTOMS
- Poor vision in one or both eyes
- Squinting or closing one eye while reading or watching tv.
- Crossed or turned eye
- Turning or tilting the head when looking at an object.
Children rarely complain of poor vision.
TREATMENT Treatment for amblyopia depends on the underlying problem. In some cases,
the strong eye is temporarily patched so the child is forced to use the weaker eye. For children with problems relating to
a refractive error, glasses may be necessary to correct vision. Problems that impair vision such as cataracts or droopy eyelids
often require surgery. Regardless of the treatment required, it is of utmost umportance that intervention is implemented as
early as possible before the child's brain learn to permanently suppress or ignore the eye.

Astigmatism
Astigmatism is the most common vision problem, but
most people, don't know what it is. This may accompany nearsightedness or farsightedness. Usually it is caused by
an irregularly shaped cornea (called corneal astigmatism). But sometimes it is the result of an irregularly shaped lens, which
is located behind the cornea; this is called lenticular astigmatism. Either kind of astigmatism can usually be corrected with
eyeglasses, contacts or refractive surgery.
SIGNS AND SYMPTOMS If you have only a small amount of astigmatism, you may not notice it or have just slightly
blurred vision. But sometimes uncorrected astigmatism can give you headaches or eyestrain, and distorted or blur your vision
at all distances. Unfortunately, astigmatism can affect anyones ability to see well in school, during sports, driving, at
work etc., so it's important to have your eyes examined at regular interval in order to detect any astigmatism early on.
WHAT CAUSES ASTIGMATISM This
occurs when the cornea is shaped more like a oblong football than a spherical basketball, which is the normal shape. In most
astigmatic eyes, the oblong or oval shape causes light rays to focus on two points in the back of your eye, rather than on
just one. This is because, like a football, as astigmatic cornea has a steeper curve and a flatter one.
TREATMENT Unless it is extreme, astigmatism can be compensated for satisfactorily with
eyeglasses or contact lenses. If your eyeglasses or contact lens prescription contains three parts rather than one, your eyecare
practitioner has found some astigmatism in one or both of your eyes.
Many patients with astigmatism believe that
they can not wear contact lenses, or that only rigid lenses (RGP) can correct astigmatism.This was true several years ago,
but not there are soft contacts that correct astigmatism, called toric.
Depending on the amount of astigmatism,
you may have to stay with prescription eyeglasses or depending on the type and severity of our astigmatism, you may also be
able to have it corrected with refractive surgery. Discuss with your eye doctor which is best for you.

Cataracts
A cataract is a clouding
of the natural lens, the part of the eye responsible for focusing light and producing clear, shape images. The lens is contained in a sealed bay or capsule. Over time, the cells accumulate causing the lens to cloud, making
images look blurred or fuzzy. For most patients, cataracts are a natural result of aging.

Diabetic
Retinopathy
This is a disease that occurs when the pancreas does not secrete
enough insulin or the body is unable to process it properly. Insulin is the hormone that regulates the level of sugar (glucose)
in the blood. Diabetes can affect children and adults.
HOW DOES DIABETES AFFECT THE
RETINA Patients with diabetes are more likely to develop eye problems such as cataracts and glaucoma,
but the disease's affect on the retina is the main threat to to vision. Most patients develop diabetic changes in the
retina after approximately 20 years. The effect of diabetes on the eye is called diabetic retinopathy.
 Over time, diabetes affects the circulatory system of the retina. The earliest phase of the disease is
known as background diabetic retinopathy. In this phase, the arteries in the retina become weakened and leak, forming small,
dot-like hemorrhages. These leaking vessels often lead to swelling or edema in the retina and decreased vision.
The next stage is known as proliferative diabetic retinopathy. In this stage, circulation problems cause
areas of the retina to become oxygen-deprived or ischemic. New, fragile, vessels develop as the circulatory system attempts
to maintain adequate oxygen levels within the retina. This is called neovascularization. Unforunately, these delicate vessels
hemorhage easily. Blood may leak into the retina and vitreous, causing spots or floaters along with decreased vision.
 In the later phases of the disease, continued abnormal vessel growth and scar tissue may cause serious
problems such as retinal detachment and glaucoma.
SIGNS AND
SYMPTOMS Affect of diabetic retinopathy on vision varies widely, depending on the
stage of the disease. Some common symptoms of diabetic retinopathy are listed below, however, diabetes may cause other eye
symptoms.
*Blurred vision (this is often linked to blood sugar levels)
*Floaters and flashes *Sudden loss
of vision 
DETECTION AND DIAGNOSIS Diabetic
patients require routine eye examinations so related eye problems can be detected and treated as early as possible. Most diabetic
patients are frequently examined by an internist or endocrinologist who in turn work closely with the eye doctor.
TREATMENT
Diabetic retinopathy is treated in many ways depending on the stage of the disease and the specific problem that
requires attention. The retinal surgeon relies on several tests to monitor the progression of the disease and to make decisions
for the appropriate treatment. These include: fluorescein angiography, retinal photography and ultrasound imaging of the eye.
The abnormal growth of tiny blood vessels and the associated complication of bleeding is one of the most common problems
treated by vitro-retinal surgeons. Laser surgery called pan retinal photocoagulation (PRP) is usually the treatment of choice
for this problem.
Vitrectomy is another surgery commonly needed for diabetic patients who suffer a vitreous hemorrhage
(bleeding in the gel-like substance that fills the center of the eye). During a vitrectomy, the retina surgeon carefully removes
blood and vitreous from the eye, and replaces it with clear salt solution (saline). At the same time, the surgeon may also
gently cut strands of vitreous attached to the retina that create traction and could lead to retinal attachment or tears.
Low Vision Devices are available for Diabetic
Retinopathy, click on our "MDE Low Vision Store" to
buy Low Vision Devices.

Epitretinal Membrane (ERM) (Macular Pucker)
ERM is a cellophane-like membrane that forms over the macular. It is typically a sow-progressing problem that affects the central vision by causing blur and distortion. As it progresses, the traction of the membrane on the macula may cause
swelling. ERM is seen most often i people over 75 of age. It usually occurs for unknown reasons, but may be associated
with certain eye diseases such as: diabetic retinopathy, posterior vitreous detachment, retinal detachment, trauma and many
others.
SIGNS AND SYMPTOMS *Blurred vision *Double vision
that is noticeable even with one eye covered *Distorted vision (straight lines may appear bent or wavy)
DETECTION AND DIAGNOSIS Your eye doctor is able to detect ERM with an ophthalmoscopy
during an examination of the retina. It has a glistening, cellophane-like appearance. The affect of ERM on the patient's
central vision is assessed with a visual acuity test and the Amsler Grid (see above in ARMD for Amsler Grid,
you can print to your computer). If the doctor suspects macular swelling, he may order fluorescein.
TREATMENT A procedure called a membrane peel is performed when vision has deteriorated
to the point that it is impairing the patient's lifestyle. Most vitreo-retinal surgeons recommend waiting for treatment
until vision has decreased to the point that the rish of the procedure justifies the improvement.
The Membrane
Peel is performed under a local anesthesia in an operating room. After making tiny incisions the membrane peel is often done
in conjunction with a procedure called vitrectomy.

Glaucoma
This is a disease caused by increased intraocular pressurer (IOP), resulting
either from a malformation or malfunction of the eye's drainage structures. Left untreated, an elevated IOP causes irreversible
damage the optic nerve and retinal fibers resulting in a progressive, permanent loss of vision. However, early detection
and treatment can slow, or even halt the progression of the disease.

cup to disc ratio TYPES OF GLAUCOMA
 Open Angle- is the most common type of glaucoma. Even though the
anterior structures of the eye appear normal, aqueous fluid builds within the anterior chamber, causing the IOP to become
elevated. Left untreated, this may result i permament damage of the optic nerve and retina.
Acute Angle Closure- Only about 10% of the population with glaucoma has this type. Abnormality
of the structures in the front of the eye. In most cases, the space between the iris and cornea is more narrow than normal,leaving
a smaller channel for the aqueous to pass through. If the flow of aqueous becomes completely blocked, the IOP rises sharply,
causing a sudden angle closure attack.
Secondary Glaucoma- Occurs
with other diseases or problems within the eye such as; inflammation, trauma, previous surgery, diabetes, tumor and certain
meds. For this type, both the glaucoma and the underlying problem must be treated.
Congenital- Rare type of glaucoma that is generally seen in infants. In most cases, surgery
is required.  Because glaucoma does not cause symptoms in most cases, those who are 40 or older
should have an annual examination including a measurement of the intraocular pressure (IOP). Those who are glaucoma suspects
may need additional testings. SIGNS
& SYMPTOMS- Angle Closure (emergency) *Sudden decrease of vision *Extreme eye
pain *Headaches *Nausea and vomiting *Glare and light sensitivity Congenital *Tearing *Light
sensitivity *Enlargement of the cornea
DETECTION AND DIAGNOSIS Glaucoma
does not cause symptoms in most cases, those who are 40 or older should have an annual examination including a measurement
of the intraocular pressure. Those who are glaucoma suspects may need additional testing. Mearsuring the intraocular pressure,
evaluate the health of the optic nerve, test the peripheral vision.
The progression of glaucoma is monitored with
a visual field test. This test maps the peripheral vision, allowing the doctor to determine the extent of vision loss form
glaucoma and a measure of the effectiveness of the treatment.
TREATMENT Patients
with glaucoma require only medication to control the eye pressure. Sometimes, several medications that complement each other
are necessary to reduce the pressure adequately. Surgery is indicated when medical treatment fails to lower the pressure
satisfactorily.
Low Vision Devices are available for Glaucoma, click on our "MDE Low
Vision Store" to buy Low Vision Devices.

Hyperopia Farsightedness
or hyperopia, occurs when light entering the eye focuses behind the retina, instead of directly on it. This is caused by a cornea that is flatter, or an eye that is shorter, than a normal eye. Farsightedness patients usually have trouble
seeing up close, but may also have difficulty seeing far away as well. Young patients with mild to moderate hyperopia
are often able to see clearly because their natural lens can adjust, or accommodate to increase the eye's focusing ability.
However, as the eye gradually loses the ability to accommodate (about 40 years of age), blurred vision from hyperopia
often becomes more apparent.
SIGNS AND SYMPTOMS *Difficulty
seeing up close *Blurred distance vision (occurs with highter amounts of hyperopia) *Eye fatigue when
reading *Eye strain *Crossed eyes in children
TREATMENT Eyeglasses or contact lenses are required. However, it also depends on several factors such as the patient's
age, activities and occupation and ability to compensate for their farsightedness with accommodation.
 Hypertensive Retinopathy
The findings in hypertensive retinopathy all stem form hypertension, induced changes to the retinal microvasculature.
Hypertension leads to a laying down of cholesterol into the tunica intima of medium and large arteries. This leads to an overall
reduction inthe lumen size of these vessels. In arteriolosclerosis, hypertension leads leads to focal closure of the retinal
microvasculature. This gives rise to microinfarcts (cotton wool spots) and superficial hemorrhages. In extreme cases, disc
edems develops. The mechanism behind this phenomenon is poorly understood, but it may be related to a hypertension- related
increase in intracranial pressure and hence is considered true papilledema. Arteriolosclerotic changes in the retinal
microvasculature persist even with the reduction of systemic blood pressure. However, hypertensive retinopathy changes
resolve over time with the reduction of systemic blood pressure (BP) Cotton Wool Spots develop 24 to 48 hours with the elevation
of BP, and resolve in tow to 10 weeks with the lowering of BP. Amacular star develops within several weeks of the development
of elevated BP and resolves within months to years after the BP in reduced. Papilledema develops within days to weeks of increased
BP and resolves within weels to months following BP lowering.
 Findings in hypertensive retinopathy include cotton wool spots and flame shaped hemorrhages. Only
rarely will there be retinal or macular edema. in advanced cases, there will be a macular star (ring of exudates from the
disc to the macular) and disc edema. Arteriolosclerosis (arteriolar narrowing, arterio-venous crossing changes with venous
constriction and banking, arteriolar color changes, vessel sclerosis) is often found concurrently.
SIGNS AND SYMPTOMS Patients with hypertensive retinopathy, as
expected, suffer form hypertension. However, the hypertension may be unknown to the patient and the eye exam may yield
the first clue to this relative asymptomatic systemic disease. Most commonly, the patient is middle age or older and more
common in African-Americans than Caucasians.
Low Vision Devices are available for Hypertensive Retinopathy, click
on our "MDE Low Vision Store" to buy Low Vision Devices.

Myopia
Nearsightedness or myopia, occurs when light entering the eye focuses in front of the retina
instead of directly on it. This is caused by a cornea that is steeper, or an eye that is longer, than a normal eye.
Nearsighed patients typically see well up close, but have difficulty seeing far away.
This
is often discovered in school-age chidren who report having trouble seeing the chalkboard. Near-sightedness usually becomes
progressively worse through adolescence and srabilizes in early adulthood. It is an inherited problem.
SIGNS AND SYMPTOMS *Blurry distance vision *Vision seems
clearer when squinting
DETECTION Myopia
is detected with a acuity test and refraction.
TREATMENT Depends on several factors such as the patient's age, activities and occupation. Vision can be
corrected with glasses, contacts or surgery. Refractive procedures such as Lasik can be considered for adults when the prescription
has remained stable for at least one year.

Ocular Histoplasmosis Syndrome
This
is caused by a fungus commonly found in the dust and soil of the Mississippi-Ohio River Valley region. Approximately 62% of the adult population living in this region are carriers. Histoplasmosis is contracted by inhaling
dust that carries the fungal spores. Its effect on the body can vary widely in severity from one person to another. Many carriers
have no symptoms at all, but those with mild exposure may experience flu-like symptoms and mild respiratory infections.
Histolplasmosis is more likely to become a serious problem in people who already have weakened immune system.
The fungus may affect the eye by causing small areas of inflammation and scarring of the retina.
These are called "histo spots" and may be found in both eyes. Their affect on vision depends on the location of
the scars. Scarring in the peripheral area of the retina may have little or o impact o vision, while a central scar affecting
the macula may cause a prominent blind spot.
SIGNS AND
SYMPTOMS *Many patients with histo spots in their eyes have no symptoms. *Others may experience
the following: *Distorted vision *Blind spots *Scars in the retina, ranging in severity
TREATMENT Usually requires no treatment except when abnormal
blood vessels develop in the central retina. For these patients, laser treatment may be necessary. In some cases, surgical
removal of the tiny abnormal vessels has been successful. Click on "MDE Low Vision
Store" to buy Low Vision products.

Posterior Capsule Opacification
Posterior
Capsule Opacificaton (PC Haze), is the remaining portion of the capsule becomes clouded in about 25% of cataract
surgery patients. When this occurs, patients pexperience symptoms similar to thoe from the original cataract. That's why
posterior capsule opacification (PC Haze) is also known as a secondary cataract.
The lens capsule is the thin, elastic-like bag that holds the intracocular lens (IOL) in
position after cataract surgery. During the operation, the front (anterior) portion of the lens capsule is carefully
opened and the cataract is removed. The IOL is inserted into the remaining (posterior) portion of the capsule.
SIGNS AND SYMPTOMS *Gradual decrease of vision *Blurred
vision *Glare from lights and sun
TREATMENT Simple procedure called the YAG posterior capsulotomy is performed to restore vision lost form the clouded capsule.
The YAG is a type of "cold" laser used to create a small opening in to center of the capsule, allowing a clear area
for light to enter the eye. The procedure is painless, requires no anesthesia and has very little risk since no incision is
required. After the dilating drops wear off from the procedure, most patients notice an immediate improvement in vision. The
improvement each person experiences is dependent on the extent of the capsular clouding and the overall health of their eyes.

Posterior Vitreous Detachment
The space between the crystalline lens and the retina is filled with a clear, gel-like substance callled
vitreous. In a newborn, the vitreous has an egg-white consistency and is firmly attached to the retina. With age, the vitreous
thins and may separate from the back of the eye. This is called posterior vitreous detachment (PVD).
As the vitreous pulls free from teh retina, it is often accompanied by light flashes or4 floaters. Floaters
are caused by tiny bits of vitreous gel or cells that cast shadows on the retina. Flashes occur when the vitreous tugs on
the sensitive retina tissue.
There are other more serious causes of flashes and floaters,
with include, retinal tears, retinal detachment, infection, inflammation, hemorrhage, or an injury such as a blow to the head
may also cause floaters and flashes. (Have you ever seen stars after bumping your head) Occasionally, flashes of light are
caused by neurological problems such as a migraine headache. When related to a headache, the flashes of light are seen in
both eyes and usually lasts 20-30 minutes before the headaches starts.
SIGNS
AND SYMPTOMS *Black spots or spider webs that seem to float in the vision in a cluster or alone. *Spots
that move or remain suspended in one place. *Flickering or flashing lights that are most prominent when looking at a
bright background like a clear sky.
SYMPTOMS THAT MAY INDICATE
A MORE SERIOUS PROBLEM *Sudden decrease of vision along with flashes and floaters *Veil or curtain
that obstructs part or all of the vision *Sudden increase in the number of floaters
Notify
your physician immediately if you notice a sudden shower of floaters, new light flashes, a veil or curtain obstructing your
vision or any other change.
TREATMENT Because
of the rish, surgery is rarely indicated for PVD except when the floaters obscure the vision. In these cases, surgical removal
of the vitreous may be considered only if the vision is significantly affected. This treatment is rarely needed since floaters
typically become less bothersome over a period of weeks to months as they settle below the line of sight. However,
vitrectomy may be indicated in a select group of patients with visually disabling vitreous floaters, as long as an objective
assessment of the patient's visual disfunction from the floaters is made.

Presbyopia
This is which
the natural lens can no longer accommodate. Accommodation is the eye's way of changing its focusing distance: the lens
thickens, increasing its ability to focus close-up. At about age 40, the lens becomes less flexible and accommodation
is gradually lost. It is a normal process that everyone eventually experiences. However, nearsighted people can simply take
their glasses off because they see best close-up.
SIGNS AND SYMPTOMS *Difficulty seeing clearly for close work *Print seems to have less contrast *Brighter or more direct
light required for reading *Reading material must be held further away to see *Fatique and eyestain when reading
TREATMENT Treatment for presbyopia is very simple, and depends
on patients age, lifestyle, occupation, and hobbies. Progressive bifocals are a great source, vision at all distances,
and you can not see the line in your eyeglasses. Trifocals, works like a progressive lens but you can see the line in
your eyeglasses, bifocal with the line, blended bifocal (on-line, but not progressive), 2 pairs of eyeglasses, one for
distance and one for reading, or bifocal contact lenses (depending on patients prescription), monovision with contacts, adjusting
one eye for distance vision, and the fellow eye for reading vision.

Free Audio Bible Material
Diabetes
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Retinitis
Pigmentosa
Retinitis Pigmentosa (RP) is a rare, inherited disease in which the
light-sensitive retina of the eye slowly and progressively degenerates. Eventually, blindness.
RP is a inherited disease, even if your mother or father do not have RP, you can still have the eye
disease when at least one parent carries an altered gene associated with the trait. Not much is known about what causes retinitis
pigmentosa.
SIGNS AND SYMPTOMS The
first signs of retinitis pigmentosa usually occurs in early childhood, when both eyes typically are affected. Night vision
can be poor, and the field of vision may begin to narrow. During later stages of RP, only a small area of central vision reamins,
along with slight peripheral vision.
TREATMENT No treatment is currently available for RP, although some practitioners believe that
vitamin A may slightly delay vision loss. Low Vision therapy may be helpful, because it is easier to adjust to declining vision
in earlier stages of vision loss. Low Vision Devices that can help magnify and illuminate objects in home or office. Click
on our "MDE Low Vision Store" to buy "Low Vision Devices"

Subconjunctival Hemorrhage
Subconjunctival hemorrhage occurs when a small blood vessel under the conjunctiva
breaks and bleeds. It may occur spontaneously or from coughing, heavy lifting or vomiting. In some cases, it may develop following
eye surgery or trauma. Subconjunctival hemorrhage tends to be more common among those with diabetes and hypertension.
While this might look frightening, it is essentially harmless. The blood becomes trapped underneath the clear conjunctival
tissue, much like a bruise. The blood is visible because it show through the thin, clear conjunctiva. The blood naturally
absorbs within one to three weeks.
SIGNS AND SYMPTOMS *Red, bloody patch on the white of the eye *Painless *No change in vision
TREATMENT It might look like an emergency, but it does not affect the vision and no treatment
is required.

Toxoplasmosis
This is a disease provoked by the obligate intracellular protozoan
Toxoplasma gondii. It is found in a variety of mammal and bird hosts. The most common intermediate host is the cat.
It is one of the most frequent causes of retinochoroiditis in humans, with more than 60% of the United States population
and up to 75% of the world's general population possessing some seropositive findings.
This disease exists
in humans in two forms: (1) actively motile tachzoites and (2) encysted Toxoplasma gondii called brachzoites. The oocysts
that contain the organisms which produce infection are excreted in fecal material and may lie dormant in the soil until ingested
by other animals, resulting in infection. Human infection may occur from ingeston of contaminated or undercooked meat anddairy
products, direct or indirect ingestion of cate feces and tranplacental transmission form an infected mother to the fetus.
Toxo can only be transmitted to a fetus during maternal parastemia. Congenital toxoplasmosis accounts for the majority of
cases encountered in clinical practice.
The inflammatory fundus lesions are composed of mononuclear cells, with a liberation of lymphocytes, macrophages, epithelioid
and plasma cells. The resulting retinal vasculitis contributes to the breakdown of the blood-retinal barrier and leads to
a compromise in retinal function, with subsequent destruction and thickening.
SIGNS AND SYMPTOMS Unilateral, mild ocular pain, blurred vision and new onset of floating
spots. Patients often describe their vision as hazy. Clinical findings may include granulomatous iritis, vitritis, optic disc
swelling, neuroretinitis, vasculitis and retinal vein occlusion in the vicinity of the inflammation, in the actively involved
eye.

Uveitis
This
is a general term that refers to inflammation or swelling of the eye's structures responsible for its blood supply. These
structures anre collectively known as the uveal tract, and include the iris, ciliary body and choroid.
Uveitis
is classified by the structures if effects, the underlying cause and whether it is chronic or acute in nature. There are four
categories of uveitis; Anterior uveitis (also known as iritis), Intermediate uveitis, affects the ciliary body, vitreous and
retina, and diffuse uveitis affects structures both in the front and back of the eye.
Common causes include, infections
and underlying diseases, but in some cases the cause is unknown.
SIGNS
AND SYMPTOMS Anterior *Light sensitivity *Blurred vision *Redness
around the iris *Pain that may range from aching or soreness to intense discomfort *Small Pupil *Tearing *Elevated IOP Intermediate *Often affects both eyes *Floaters *Blurred
vision Posterior *Blurred vision *Pain (if optic nerve is involved)
TREATMENT Treatment for uveitis is
dependent on the severity of the disease and the ocular structures involved. Topical eye drops and/or oral medications are
prescribed to reduce inflammation. In some cases, meds are required to lower the IOP.
American Medical Association
Shriner's Hospital
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